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Desmoglein 2 is associated with dihydroartemisinin resistance in HeLa cervical cancer cells: a potential link to the PI3K/AKT signaling pathway

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Published in:Discover Oncology
Format: Online Article RSS Article
Published: 2026
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container_title Discover Oncology
description
discipline_display Endocrinology
discipline_facet Endocrinology
format Online Article
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genre Journal Article
id rss_article:90558
institution FRELIP
journal_source_facet Discover Oncology
last_indexed 2026-06-12T02:00:45.559Z
publishDate 2026
publishDateSort 2026
record_format rss_article
spellingShingle Desmoglein 2 is associated with dihydroartemisinin resistance in HeLa cervical cancer cells: a potential link to the PI3K/AKT signaling pathway
Endocrinology
General
Endocrinology
sub_discipline_display General
sub_discipline_facet General
subject_display Endocrinology
General
Endocrinology
Endocrinology
General
Endocrinology
subject_facet Endocrinology
General
Endocrinology
title Desmoglein 2 is associated with dihydroartemisinin resistance in HeLa cervical cancer cells: a potential link to the PI3K/AKT signaling pathway
title_auth Desmoglein 2 is associated with dihydroartemisinin resistance in HeLa cervical cancer cells: a potential link to the PI3K/AKT signaling pathway
title_full Desmoglein 2 is associated with dihydroartemisinin resistance in HeLa cervical cancer cells: a potential link to the PI3K/AKT signaling pathway
title_fullStr Desmoglein 2 is associated with dihydroartemisinin resistance in HeLa cervical cancer cells: a potential link to the PI3K/AKT signaling pathway
title_full_unstemmed Desmoglein 2 is associated with dihydroartemisinin resistance in HeLa cervical cancer cells: a potential link to the PI3K/AKT signaling pathway
title_short Desmoglein 2 is associated with dihydroartemisinin resistance in HeLa cervical cancer cells: a potential link to the PI3K/AKT signaling pathway
title_sort desmoglein 2 is associated with dihydroartemisinin resistance in hela cervical cancer cells: a potential link to the pi3k/akt signaling pathway
topic Endocrinology
General
Endocrinology
url https://link.springer.com/article/10.1007/s12672-026-05425-y