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Chrysin alleviates pressure overload–induced myocardial remodeling through regulating the PI3K/AKT/NRF2 pathway–mediated oxidative stress response

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Published in:Animal Models and Experimental Medicine
Format: Online Article RSS Article
Published: 2026
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container_title Animal Models and Experimental Medicine
description
discipline_display Laboratory and Experimental Medicine
discipline_facet Laboratory and Experimental Medicine
format Online Article
RSS Article
genre Journal Article
id rss_article:91356
institution FRELIP
journal_source_facet Animal Models and Experimental Medicine
last_indexed 2026-06-20T21:44:45.448Z
publishDate 2026
publishDateSort 2026
record_format rss_article
spellingShingle Chrysin alleviates pressure overload–induced myocardial remodeling through regulating the PI3K/AKT/NRF2 pathway–mediated oxidative stress response
Laboratory and Experimental Medicine
General
Laboratory and Experimental Medicine
sub_discipline_display General
sub_discipline_facet General
subject_display Laboratory and Experimental Medicine
General
Laboratory and Experimental Medicine
subject_facet Laboratory and Experimental Medicine
General
Laboratory and Experimental Medicine
title Chrysin alleviates pressure overload–induced myocardial remodeling through regulating the PI3K/AKT/NRF2 pathway–mediated oxidative stress response
title_alt La crisina alivia la remodelación miocárdica inducida por sobrecarga de presión mediante la regulación de la vía de respuesta al estrés oxidativo PI3K/AKT/NRF2
La chrysine atténue le remodelage myocardique induit par la surcharge de pression via la régulation de la voie PI3K/AKT/NRF2 médiant la réponse au stress oxydatif
Crisina alivia a remodelação miocárdica induzida por sobrecarga de pressão através da regulação da resposta ao estresse oxidativo mediada pela via PI3K/AKT/NRF2
title_auth Chrysin alleviates pressure overload–induced myocardial remodeling through regulating the PI3K/AKT/NRF2 pathway–mediated oxidative stress response
title_es_txt La crisina alivia la remodelación miocárdica inducida por sobrecarga de presión mediante la regulación de la vía de respuesta al estrés oxidativo PI3K/AKT/NRF2
title_fr_txt La chrysine atténue le remodelage myocardique induit par la surcharge de pression via la régulation de la voie PI3K/AKT/NRF2 médiant la réponse au stress oxydatif
title_full Chrysin alleviates pressure overload–induced myocardial remodeling through regulating the PI3K/AKT/NRF2 pathway–mediated oxidative stress response
title_fullStr Chrysin alleviates pressure overload–induced myocardial remodeling through regulating the PI3K/AKT/NRF2 pathway–mediated oxidative stress response
title_full_unstemmed Chrysin alleviates pressure overload–induced myocardial remodeling through regulating the PI3K/AKT/NRF2 pathway–mediated oxidative stress response
title_pt_txt Crisina alivia a remodelação miocárdica induzida por sobrecarga de pressão através da regulação da resposta ao estresse oxidativo mediada pela via PI3K/AKT/NRF2
title_short Chrysin alleviates pressure overload–induced myocardial remodeling through regulating the PI3K/AKT/NRF2 pathway–mediated oxidative stress response
title_sort chrysin alleviates pressure overload–induced myocardial remodeling through regulating the pi3k/akt/nrf2 pathway–mediated oxidative stress response
topic Laboratory and Experimental Medicine
General
Laboratory and Experimental Medicine
url https://onlinelibrary.wiley.com/doi/10.1002/ame2.70230?af=R